金水缓纤组分方阻抑成纤维细胞活化和巨噬细胞M2极化改善博来霉素诱导大鼠模型肺纤维化

基于组分配伍研究形成的金水缓纤组分方，从成纤维细胞活化、巨噬细胞M2极化角度探讨了金水缓纤组分方干预博来霉素诱导的肺纤维化模型大鼠作用机制。

Zhao, P., et al., Effective-compound combination inhibits the M2-like polarization of macrophages and attenuates the development of pulmonary fibrosis by increasing autophagy through mTOR signaling. International Immunopharmacology, 2021. 101: p. 108360.